Primary Recipient

UNIVERSITY OF ARKANSAS SYSTEM

Amount

$398,750.00

Award Date

5/15/2009

Administering Agency

Health and Human Services, Department of / National Institutes of Health

CDFA Number

93.701

CDFA Program Title

Trans-NIH Recovery Act Research Support

Description

Regulation of caspase-1 by Sod2 in the heart? Apoptosis of cardiomyocytes is increased in human heart failure, and several studies have subsequently suggested that cardiomyocyte apoptosis is a central mechanism in heart failure. The exploratory studies